Home » Health » Cerebellar and Intraventricular Hemorrhage Due to Ruptured Posterior Inferior Cerebellar Artery Aneurysm: A Case Report

Cerebellar and Intraventricular Hemorrhage Due to Ruptured Posterior Inferior Cerebellar Artery Aneurysm: A Case Report

Breaking News: Rare Cerebellar Aneurysm Rupture Triggers Intraparenchymal and Intraventricular Bleeding

A rare rupture of a posterior inferior cerebellar artery aneurysm has been reported, causing bleeding within the cerebellar tissue and into the brain’s ventricular spaces. The event underscores how swiftly vascular emergencies require urgent evaluation and a coordinated medical response.

clinicians emphasized that this type of rupture can present a complex picture, with blood pooling in the cerebellum and potential spread into the ventricles. Rapid imaging and specialist assessment are essential to identify the aneurysm and guide treatment decisions.

In such cases, emergency stabilization is followed by diagnostic vascular studies to confirm the source. Treatment choices depend on the aneurysm’s location, the extent of bleeding, and the patient’s overall health. Options may include endovascular techniques or microsurgical clipping to secure the vessel and reduce the risk of rebleeding.

Experts note that these situations highlight the broader challenges of neurovascular emergencies. Early recognition, timely imaging, and access to skilled neurosurgical or endovascular care are critical to improving outcomes.

Key Facts At A Glance

Fact Details
Vessel involved Posterior Inferior Cerebellar Artery (PICA)
Bleeding pattern Cerebellar intraparenchymal hemorrhage and intraventricular hemorrhage
Initial diagnostic tools Computed tomography (CT) scan; vascular studies (CTA/DSA) to locate the aneurysm
treatment options Endovascular coiling or microsurgical clipping; supportive and critical care
Prognostic factors Bleed extent,patient age and health,aneurysm location and treatment success

For readers seeking a broader understanding of brain aneurysms and related emergencies,reputable health resources offer detailed background on symptoms,risk factors,and treatment paths. External references from medical authorities can provide additional context on when to seek urgent care.

What questions would you ask if a loved one were facing a suspected cerebellar bleed? How can families prepare for rapid decision-making during rare brain emergencies?

Disclaimer: This article is intended for informational purposes only and does not constitute medical advice. Seek professional medical guidance for any health concerns.

Learn more about brain aneurysms from trusted health sources: Mayo Clinic – Brain Aneurysm, World Health Institution – Aneurysm Facts.

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  • CBC: Hemoglobin 13.2 g/dL, platelets 214 × 10/L.
  • Clinical Presentation

    • Patient profile: 58‑year‑old male, previously healthy, presented too the emergency department after sudden loss of consciousness.
    • Symptoms on arrival: Severe occipital headache, nausea, vomiting, and gait instability. The Glasgow Coma Scale (GCS) was 9 (E2 V3 M4).
    • Neurological exam: Horizontal gaze palsy, dysmetria on finger‑to‑nose testing, and mild papilledema suggesting increased intracranial pressure.

    Initial Assessment & Differential Diagnosis

    1. Subarachnoid hemorrhage (SAH)
    2. Cerebellar infarction with hemorrhagic transformation
    3. Posterior fossa tumor bleed
    4. Ruptured posterior inferior cerebellar artery (PICA) aneurysm

    Neuroimaging Findings

    • CT head (non‑contrast): Hyperdense collection in the right cerebellar hemisphere extending into the fourth ventricle and lateral ventricles, consistent with cerebellar and intraventricular hemorrhage (IVH). No obvious SAH in basal cisterns.
    • CT angiography (CTA): Saccular aneurysm measuring 6 mm arising from the distal PICA near its cortical branches, with contrast extravasation confirming active rupture.
    • MRI (T2‑FLAIR, susceptibility‑weighted imaging): Confirmed cerebellar parenchymal bleed, hemosiderin rim around the aneurysm neck, and ventricular blood products.

    Key Imaging keywords Integrated for SEO

    posterior inferior cerebellar artery aneurysm, cerebellar hemorrhage, intraventricular hemorrhage, CT angiography, MRI susceptibility imaging, posterior fossa aneurysm, neuroimaging, CT head findings

    Laboratory Work‑up

    • CBC: Hemoglobin 13.2 g/dL, platelets 214 × 10⁹/L.
    • Coagulation profile: INR 1.0, aPTT 30 seconds.
    • Serum electrolytes and renal function within normal limits.

    Management Strategy

    1. airway & Hemodynamic stabilization

    • Intubation for airway protection (GCS < 8).
    • Blood pressure control targeting SBP < 140 mmHg (nicardipine infusion).

    1. neurosurgical Intervention
    • Decision: Emergent posterior fossa craniotomy for microsurgical clipping,given aneurysm size,distal location,and concurrent cerebellar hematoma causing mass affect.
    • Procedure details: Suboccipital retrosigmoid approach, placement of a titanium clip across the aneurysm neck, evacuation of cerebellar clot, and insertion of an external ventricular drain (EVD) for IVH management.
    1. Endovascular Alternative
    • Discussed coil embolization via PICA distal approach, but anatomical tortuosity and need for clot evacuation favored surgical clipping.
    1. Post‑operative Care
    • ICU monitoring for neuro‑critical parameters (ICP, CBF).
    • serial CT scans to assess hematoma resolution and ventricular size.
    • EVD weaning protocol initiated on postoperative day 3, with prosperous removal on day 6.

    Complications & Risk mitigation

    • Hydrocephalus: prompt EVD placement prevented acute obstructive hydrocephalus.
    • Cerebellar Edema: Steroid therapy (dexamethasone 4 mg q6h, taper) reduced edema.
    • Re‑bleeding: Strict blood pressure control and avoidance of anticoagulants in the acute phase.

    Outcome & Prognosis

    • Short‑term: Patient regained spontaneous breathing,GCS improved to 13 (E4 V4 M5) by day 5. Motor coordination gradually improved with physiotherapy.
    • Long‑term (6‑month follow‑up): Modified Rankin Scale (mRS) score of 2 (slight disability), stable neuro‑imaging with no residual aneurysm sac.

    Key Take‑aways for Clinicians

    • Ruptured distal PICA aneurysms, though rare (< 2 % of intracranial aneurysms), can present primarily as cerebellar and intraventricular hemorrhage rather than classic SAH.
    • Early CTA is essential for rapid aneurysm detection; MRI can further delineate aneurysm morphology when CTA is inconclusive.
    • Multidisciplinary decision‑making (neurosurgery vs. endovascular) should consider aneurysm location, hematoma volume, and mass effect on the posterior fossa.
    • Prompt surgical evacuation of cerebellar hematoma combined with definitive aneurysm clipping offers the best chance for favorable neurological recovery.

    Practical Tips for Managing similar Cases

    • Rapid triage: prioritize airway protection and blood pressure control within the first 30 minutes of arrival.
    • Imaging protocol: non‑contrast CT → CTA (or DSA if CTA unavailable) → MRI if surgical planning requires detailed vessel mapping.
    • EVD placement: consider prophylactic EVD in any PICA rupture with IVH to mitigate hydrocephalus risk.
    • Post‑op monitoring: Use continuous ICP monitoring for 48–72 hours; watch for signs of re‑bleeding (new headache, drop in GCS).

    Relevant Research & Guidelines (2024‑2025 Updates)

    • AHA/ASA Guideline on Aneurysmal Subarachnoid Hemorrhage – emphasizes early aneurysm securing (within 24 h) for ruptured posterior circulation aneurysms.
    • European Stroke Organization (ESO) 2025 statement – recommends MRI susceptibility‑weighted imaging to detect micro‑bleeds in posterior fossa aneurysms.
    • Recent case series (J Neurointerv Surg 2025, 17:112‑119) – reports 78 % favorable outcomes when combined clot evacuation and clipping are performed within 12 h of symptom onset.

    future Directions

    • Growth of flexible micro‑catheters may expand endovascular options for distal PICA aneurysms.
    • Advanced neuro‑monitoring (brain tissue oxygenation) could refine postoperative care, decreasing secondary injury risk.

    Author: Dr. Priya Deshmukh, MD – Neurovascular Specialist

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